![]() ![]() We report a method for the speciation of submicrogram per liter levels of arsenic in water. This strengthens the hypothesis that 18-hydroxydeoxycorticosterone contributes to the etiology of hypertension, possibly by a mechanism involving stressinduced release of adrenocorticotropic hormone.Ībstract: Speciation of arsenic is crucial for assessing health implications from arsenic ingestion and for effective removal of arsenic from water. ![]() Daily subcutaneous injections of 200 micrograms, a low physiological dose, significantly increase the blood pressure of unilaterally nephrectomized saline-treated rats after 2 weeks. Direct proof that 18-hydroxydeoxycorticosterone is capable of causing hypertension is present. Its production is abnormally high in some forms of hypertension in man and rat. TL DR: Daily subcutaneous injections of 200 micrograms, a low physiological dose, significantly increase the blood pressure of unilaterally nephrectomized saline-treated rats after 2 weeks, which strengthens the hypothesis that 18-hydroxydeoxycorticosterone contributes to the etiology of hypertension, possibly by a mechanism involving stressinduced release of adrenocorticotropic hormone.Ībstract: 18-Hydroxydeoxycorticosterone is an adrenal steroid hormone causing salt and water retention and is secreted in greatly increased amounts in response to the pituitary hormone adrenocorticotropic hormone.
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